ESA-SRB-AOTA 2019

Differential effects of activin A on the responses to inflammatory activation in cultures of bone marrow-derived and cell line macrophages (#660)

Julia Bender 1 , Yalong Yang 1 , Monika Fijak 1 , Kate Loveland 2 3 , Sudhanshu Bhushan 1 , Mark Hedger 2 3 , Andreas Meinhardt 1 2 3
  1. Institute of Anatomy and Cell Biology, Justus-Liebig-University of Giessen, Giessen, Hesse, Germany
  2. Centre for Reproductive Health, Hudson Institute of Medical Research, Clayton, Victoria, Australia
  3. Department of Molecular and Translational Sciences, School of Clinical Sciences, Monash University, Clayton, Victoria, Australia

Testicular macrophages contribute to the immune privilege of the testis through their immunoregulatory properties. The mechanisms of regulation of the essentially anti-inflammatory testicular macrophage phenotype are not well understood, but attention has been drawn to the role of the immunoregulatory cytokine, activin A. Within the immune system, activins exert both pro- and anti-inflammatory actions, depending on the immunological context. Activin A is produced by a number of testicular cell types, including the Sertoli cells, which have been shown to possess graft-protecting activities in co-transplantation studies. In order to investigate macrophage responses to activin, two murine macrophage models were compared: CSF1-matured bone marrow-derived macrophages (BMDM) and the RAW246.7 (RAW) macrophage cell line, which differ significantly in their activation status and downstream inflammatory signalling pathways. In BMDM cultures, activin A (50 ng/ml) significantly induced the secretion of the pro-inflammatory cytokine TNF upon stimulation (6 and 24h) with bacterial lipopolysaccharide (10-100 ng/ml). The secretion of the anti-inflammatory cytokine, IL-10, upon LPS challenge was significantly decreased by activin A. Additionally, the mRNA expression of pro-inflammatory markers (Tnf, Gpr18) were significantly increased by activin A in both resting and LPS-activated BMDM, while the expression levels of anti-inflammatory markers (Chil3, Mrc1) were decreased. In contrast, production of TNF was significantly decreased by activin A in RAW macrophages. A reduction in production of the pro-inflammatory mediator, NO, was observed in both macrophage types, while its anti-inflammatory counterpart, Arg1, was elevated. In summary, these studies indicate that activin A exerts predominantly pro-inflammatory effects in BMDM, whereas preliminary data in RAW macrophages suggest an anti-inflammatory response towards activin A treatment. Further investigation of the differences between the responses of macrophages under different activation states will help to elucidate the role that activin A plays in regulating macrophage activity in the testis and other tissues.