ESA-SRB-AOTA 2019

Prevalence of primary aldosteronism in Australian primary care (#564)

Renata Libianto 1 , Morag J Young 1 , Jimmy Shen 1 2 , Grant Russell 3 , Michael Stowasser 4 , Stella Gwini 5 6 , Peta Nuttall 1 , Peter J Fuller 1 2 , Jun Yang 1 2 7
  1. Endocrine Hypertension Group, Hudson Institute of Medical Research, Clayton, VIC, Australia
  2. Endocrinology, Monash Health, Clayton, Victoria, Australia
  3. Department of General Practice, Monash University, Clayton, Victoria, Australia
  4. Faculty of Medicine, University of Queensland, St Lucia, QLD, Australia
  5. Department of Epidemiology and Preventive Medicine, Monash University, Clayton, Victoria, Australia
  6. University Hospital Geelong, Barwon Health, Geelong, Victoria, Australia
  7. Department of Medicine, Monash University, Clayton, VIC, Australia

Background: Primary aldosteronism (PA) is the most common endocrine cause of hypertension with a reported prevalence of 3 – 13% in primary care. Timely diagnosis is important, since PA carries a worse prognosis compared to blood pressure-matched essential hypertension. However, even though effective targeted treatments are available, evidence suggests that PA is substantially under-diagnosed.  A recent survey of Victorian primary care clinics revealed that < 0.1% of 7000+ hypertensive patients had a diagnosis of PA. This study is the first in Australia to establish the prevalence of PA in primary care populations.  

Methods: Twenty primary care clinics across rural and urban Victoria were invited to screen patients with newly diagnosed, untreated hypertension, for PA by measuring their aldosterone-to-renin ratio (ARR). Those with ARR > 70 pmol/mU underwent confirmatory testing with a recumbent saline infusion test. Plasma aldosterone concentration (PAC) >140 pmol/L after the infusion of 2L normal saline confirmed the diagnosis.

Results: Of 156 patients screened, 46 had an ARR > 70. Of the 27 undertaking the saline infusion test, 23 had PAC > 140 pmol/L post saline infusion, leading to a prevalence estimate of 25%. If the most stringent cut-off for post-saline PAC is used (> 280 pmol/L), 6 patients would have PA, leading to a prevalence estimate of 7%. Baseline characteristics, in particular, age, blood pressure, and potassium levels did not discriminate between those with and without PA.

Conclusions: PA is under-diagnosed in Australian primary care, and screening has identified a prevalence of up to 25% amongst patients with newly diagnosed, treatment-naïve hypertension. Whilst the prevalence is highly dependent on the diagnostic criteria, it is much higher than currently observed. Increased awareness and access to a streamlined care pathway are needed to improve the diagnostic rate of this potentially curable cause of hypertension.